AIDS: backto a destruction or a functional

AIDS: backto a destruction or a functional

AIDS: A U.S.- Made Monster?PREFACEIn an extensive article in the Summer-Autumn 1990 issue of “Top Secret”, Prof J.Segal and Dr.

L. Segal outline their theory that AIDS is a man-made disease,originating at Pentagon bacteriological warfare labs at Fort Detrick, Maryland.”Top Secret” is the international edition of the German magazine Geheim and isconsidered by many to be a sister publication to the American Covert ActionInformation Bulletin (CAIB). In fact, Top Secret carries the Naming Names column,which CAIB is prevented from doing by the American government, and which namesCIA agents in different locations in the world. The article, named “AIDS: US-Made Monster” and subtitled “AIDS – its Nature and its Origins,” is lengthy, hasa lot of professional terminology and is dotted with footnotes.AIDS FACTS”The fatal weakening of the immune system which has given AIDS its name(Acquired Immuno-Deficiency Syndrome),” write the Segals, “has been traced backto a destruction or a functional failure of the T4-lymphocytes, also called’helper cells’, which play a regulatory role in the production of antibodies inthe immune system.” In the course of the illness, the number of functional T4-cells is reduced greatly so that new anti-bodies cannot be produced and thedefenceless patient remains exposed to a range of infections that under othercircumstances would have been harmless.

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Most AIDS patients die fromopportunistic infections rather than from the AIDS virus itself. The initialinfection is characterized by diarrhea, erysipelas and intermittent fever. Anapparent recovery follows after 2-3 weeks, and in many cases the patient remainswithout symptoms and functions normally for years. Occasionally a swelling ofthe lymph glands, which does not affect the patient’s well-being, can beobserved.After several years, the pre-AIDS stage, known as ARC (Aids- Related Complex)sets in. This stage includes disorders in the digestive tract, kidneys and lungs.

In most cases it develops into full-blown AIDS in about a year, at which pointopportunistic illnesses occur. Parallel to this syndrome, disorders in variousorgan systems occur, the most severe in the brain, the symptoms of which rangefrom motoric disorders to severe dementia and death. This set of symptoms, saythe Segals, is identical in every detail with the Visna sickness which occurs insheep, mainly in Iceland. (Visna means tiredness in Icelandic). However, thevisna virus is not pathogenic for human beings. The Segals note that despite thefact that AIDS is transmitted only through sexual intercourse, bloodtransfusions and non- sterile hypodermic needles, the infection has spreaddramatically. During the first few years after its discovery, the number of AIDSpatients doubled every six months, and is still doubling every 12 months nowthough numerous measures have been taken against it.

Based on these figures, itis estimated that in the US, which had 120,000 cases of AIDS at the end of 1988,900,000 people will have AIDS or will have died of it by the end of 1991. It isalso estimated that the number of people infected is at least ten times thenumber of those suffering from an acute case of AIDS. That in the year 1995there will be between 10-14 million cases of AIDS and an additional 100 millionpeople infected, 80 percent of them in the US, while a possible vaccination willnot be available before 1995 by the most optimistic estimates. Even when suchvaccination becomes available, it will not help those already infected.

Theseand following figures have been reached at by several different mainstreamsources, such as the US Surgeon General and the Chief of the medical services ofthe US Army.”AIDS does not merely bring certain dangers with it; it is clearly a programmedcatastrophe for the human race, whose magnitude is comparable only with that ofa nuclear war”, say the Segals. ” They later explain what they mean by”programmed,” showing that the virus was produced by humans, namely Dr. RobertGallo of the Bethesda Cancer Research Center in Maryland. When proceeding toprove their claims, the Segals are careful to note that: “We have givenpreference to the investigative results of highly renowned laboratories, whoseobjective contents cannot be doubted. We must emphasize, in this connection,that we do not know of any findings that have been published in professionaljournals that contradict our hypotheses.

“DISCOVERING AIDSThe first KNOWN cases of AIDS occurred in New York in 1979. The first DESCRIBEDcases were in California in 1979. The virus was isolated in Paris in May 1983,taken from a French homosexual who had returned home ill from a trip to the EastCoast of the US.

One year later, Robert Gallo and his co-workers at the BethesdaCancer Research Center published their discovery of the same virus, which iscytotoxic. ( i.e poisonous to cells ) Shortly after publishing his discovery,Gallo stated to newspapers that the virus had developed by a natural processfrom the Human Adult Leukemia virus, HTLV-1, which he had previously discovered.However, this claim was not published in professional publications, and soonafter, Alizon and Montagnier, two researchers of the Pasteur Institute in Parispublished charts of HTLV-1 and HIV, showing that the viruses had basicallydifferent structures. They also declared categorically that they knew of nonatural process by which one of these two forms could have evolved into theother.

According to the professional “science” magazine, the fall 1984 annualmeeting of the American Association for the Advancement of Science (AAAS), wasalmost entirely devoted to the question of: to what extent new pathogenic agentscould be produced via human manipulation of genes. According to the Segals, AIDSwas practically the sole topic of discussion.THE AIDS VIRUSThe Segals discuss the findings of Gonda et al, who compared the HIV, visna andother closely-related viruses and found that the visna virus is the most similarto HIV. The two were, in fact, 60% identical in 1986. According to findings ofthe Hahn group, the mutation rate of the HIV virus was about a million timeshigher than that of similar viruses, and that on the average a 10% alterationtook place every two years. That would mean that in 1984, the difference betweenHIV and visna would have been only 30%, in 1982- 20%, 10% in 1980 and zero in1978. “This means,” say the Segals, “that at this time visna viruses changedinto HIV, receiving at the same time the ability to become parasites in humanT4-cells and the high genetic instability that is not known in otherretroviruses.

This is also consistent with the fact that the first cases of AIDSappeared about one year later, in the spring of 1979.” “In his comparison of thegenomes of visna and HIV,” add the Segals, “Coffin hit upon a remarkable feature.The env (envelope) area of the HIV genome, which encodes the envelope proteinswhich help the virus to attach itself to the host cell, is about 300 nucleotideslonger than the same area in visna. This behaviour suggests that an additionalpiece has been inserted into the genomes of the visna virus, a piece that altersthe envelope proteins and enables them to bind themselves to the T4-receptors.BUT THIS SECTION BEHAVES LIKE A BIOLOGICALLY ALIEN BODY, which does not matchthe rest of the system biochemically.The above mentioned work by Gonda et al shows that the HIV virus has a sectionof about 300 nucleotides, which does not exist in the visna virus. That lengthcorresponds with what Coffin described.

That section is particularly unstable,which indicates that it is an alien object. According to the Segals, it”originates in an HTLV-1 genome, (discovered by Gallo-ED) for the likelihood ofan accidental occurrence in HIV of a genome sequence 60% identical with asection of the HTLV-1 that is 300 nucleotides in length is zero.” Since thevisna virus is incapable of attaching itself to human T4 receptors, it must havebeen the transfer of the HTLV-1 genome section which gave visna the capabilityto do so. In other words, the addition of HTLV-1 to visna made the HIV virus.

Inaddition, the high mutation rate of the HIV genome has been explained by anotherscientific team, Chandra et al, by the fact that it is “a combination of twogenome parts which are alien to each other BY ARTIFICIAL MEANS rather than by anatural process of evolution, because this process would have immediatelyeliminated, through natural selection, systems that areScience

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